Hepatic Detoxification of Bisphenol A is Retinoid-Dependent
Oral supplementation with retinyl acetate restored phase I and phase II enzyme activities, but accelerated BPA-induced oxidative damage through enhancement of non-mitochondrial ROS production. Thus, the activities of the enzymes involved in the hepatic elimination of BPA require hepatic retinoid stores. The extent of hepatic damage that arises from acute BPA intoxication is directly affected by retinoid administration during the period of BPA exposure and hepatic retinoid stores that have accumulated over the lifetime of the organism.
eat your liver and you wont die
hopefully
I might try liver again after being on a little vegetarian experiment stuffing my face with beans and rice.
but then the liver I buy is packed in plastic
fun
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